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Host cell resistance to CagA translocation is as variable as Helicobacter pylori
2017
Matters
Since its discovery in the human stomach, the bacterium Helicobacter pylori has been branded as the cause of gastric diseases. This association is linked to the oncogenic toxin CagA produced by certain H. pylori strains, which causes severe damages but needs to be injected into the host cells to exert its toxic effect. Injection is achieved by a special bacterial transport mechanism, the Cag Type IV secretion system (Cag-T4SS). However, in nature, not all H. pylori strains infecting a patient
doi:10.19185/matters.201706000006
fatcat:uaokncb7h5hezlrwf64xegi4yu