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AngII infusion caused two-fold increase in ROS production of WT hearts (p<0.05) (but not p47 phox KO mice), which was inhibited significantly by diphenyleneiodonium (DPI, a flavoprotein inhibitor) or superoxide dismutase, significantly but slightly by NG-nitro-l-arginine methyl ester (L-NAME, a nitric oxide synthase inhibitor), but not by rotenone (mitochondrial respiratory chain inhibitor) or oxypurinol (xanthine oxidase inhibitor). Increased ROS production in WT AngII-infused hearts wasdoi:10.1136/heartjnl-2017-311726.217 fatcat:n7teja6qnfhvzhb2j4lvp7ktqa