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Receptor activator of NF-κB ligand (RANKL) triggers the differentiation of bone marrow-derived monocyte/macrophage precursor cells (BMMs) of hematopoietic origin into osteoclasts through the activation of mitogen-activated protein (MAP) kinases and transcription factors. Recently, reactive oxygen species (ROS) and antioxidant enzymes were shown to be closely associated with RANKL-mediated osteoclast differentiation. Although glutare-doxin2 (Glrx2) plays a role in cellular redox homeostasis, itsdoi:10.5483/bmbrep.2012.45.3.171 pmid:22449704 fatcat:kgc2hkcvxjaujolojzmjec6ija