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The human ABO(H) blood phenotypes arise from the evolutionarily oldest genetic system found in primate populations, but arise in critical molecular complementarity with distinct eukaryotic and prokaryotic pathogens, while the development of non-O(H) phenotypes is associated with the impaired formation of adaptive and innate immunoglobulin specificities. Indeed, compared with individuals with blood group O(H), individuals with blood group A not only have significantly higher risk of developingdoi:10.6084/m9.figshare.4714618.v282 fatcat:plcbdp7rdzcbddmmhqxhojrjyu