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Antiapoptotic but Not Antiviral Function of Humanbcl-2 Assists Establishment of Japanese Encephalitis Virus Persistence in Cultured Cells
1998
Journal of Virology
Upon infection of Japanese encephalitis virus (JEV), baby hamster kidney (BHK-21) and Chinese hamster ovary (CHO) cells were killed by a mechanism involved in apoptosis. While readily established in a variety of cell lines, JEV persistence has never been successfully instituted in BHK-21 and CHO cells. Since stable expression of humanbcl-2 in BHK-21 cells has been shown to delay JEV-induced apoptosis, in this study we investigated whether JEV persistence could be established in such cells. When
doi:10.1128/jvi.72.12.9844-9854.1998
fatcat:qcuor5lgh5c5pcmz2wj54wdpzu