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Nitric Oxide and Posttranslational Modification of the Vascular Proteome: S-Nitrosation of Reactive Thiols
Arteriosclerosis, Thrombosis and Vascular Biology
Nitric oxide (NO⅐) is known to exert its effects via guanylyl cyclase and cyclic GMP-dependent pathways and by cyclic GMP-independent pathways, including the posttranslational modification of proteins. Much ongoing research is focused on defining the mechanisms of NO⅐-mediated protein modification, the identity and function of the modified proteins, and the significance of these changes in health and disease. S-nitrosation or thionitrite formation has only been found on a limited number ofdoi:10.1161/01.atv.0000217632.98717.a0 pmid:16543494 fatcat:dhh5jfplhzhujham2pfqvbr3wq