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Pathogenesis of the hyperlipidemia of Gram-negative bacterial sepsis may involve pathomorphological changes in liver sinusoidal endothelial cells
International Journal of Infectious Diseases
Keywords: Liver sinusoidal endothelial cell fenestrations Transplantation Pseudomonas aeruginosa Pyocyanin Oxidative stress Electron microscopy Immunohistochemistry The Gram-negative bacterium Pseudomonas aeruginosa is one of the most common opportunistic pathogens, especially after liver transplantation. Pathophysiological alterations of liver sinusoidal endothelial cells (LSECs) have far-reaching repercussions on the liver and on metabolism. LSECs are perforated with fenestrations, pores thatdoi:10.1016/j.ijid.2010.02.2263 pmid:20609608 fatcat:s5hvb5ov5rhibol6ul2qr7yydu