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The purpose of this investigation is to identify hereditary, immunological, metabolomic and proteomic biomarkers for the advancement of islet autoimmunity and progression to diabetes type-1 in an expected high-danger companion. Authors examined 68 offspring: 46 who advanced AI (22/46 progressed to diabetes) and 26 coordinated controls for gender and age. Biomarkers were studied along four time axes: the most readily available example only before AI, shortly after AI, and only before the onsetdoi:10.5281/zenodo.3741997 fatcat:easpp6jkqrhspcnlm5q4lakvdi