A copy of this work was available on the public web and has been preserved in the Wayback Machine. The capture dates from 2018; you can also visit the original URL.
The file type is
Role of Nitric Oxide in the Regulation of Superoxide Dismutase and Prostaglandin F2α Production in Bovine Luteal Endothelial Cells
Journal of reproduction and development
Prostaglandin F2α (PGF) induces a rapid reduction in progesterone production (functional luteolysis) followed by tissue degeneration and cell death (structural luteolysis). Reactive oxygen species (ROS) including nitric oxide (NO) play crucial roles in the luteolytic action of PGF. The local concentration of intraluteal ROS is controlled by superoxide dismutase (SOD), the main enzyme involved in the control of intraluteal ROS. To clarify the roles of NO in the regulation of SOD in luteolysis,doi:10.1262/jrd.10-013k pmid:20519832 fatcat:2c4ixpqxangvfay2jltbl3iqzy