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Calcium/Calmodulin-Stimulatable Adenylyl cyclases are required for the potentiating effect of acute glucocorticoid exposure in the hippocampal synapse
Experience-dependent synaptic plasticity is important for learning and memory and regulated by the functions of N-methyl-D-aspartate (NMDA) and α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) glutamate receptors. The stress hormone glucocorticoid, exerts effects on synaptic plasticity through functional modification of these receptors. In this study, we have investigated the mechanisms underlying synaptic potentiation during acute stress and glucocorticoid treatment. We report thatdoi:10.21203/rs.3.rs-244813/v1 fatcat:kclid6a3cfeubhwulfayh5qguu