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Atopic dermatitis (AD) is a common skin disease associated with a T H 2 response and increased levels of T H 2-associated cytokines and IgE. The mechanisms resulting in skewing the immune response in a T H 2 direction in AD are not fully elucidated. However, such skewing has recently been associated with IL-25 in a murine model for allergic airway disease. The aim of this study was to investigate whether IL-25 may have a role in AD. We have identified IL-25-producing cells within the dermis ofdoi:10.1038/jid.2010.277 pmid:20861853 fatcat:5pju5vgkzbamzm4aomihk226ie