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Treating to target: a strategy to cure gout
Acute gout attacks and the long-term complications of gout are associated with the deposition of monosodium urate (MSU) monohydrate crystals in the joints and soft tissues, causing acute and chronic inflammation. The aim of long-term treatment is to reduce the serum urate (sUA) level to 6 mg/dl (4360 mol/l), below the saturation point of MSU, so that new crystals cannot form and existing crystals are dissolved. Serial joint aspiration studies confirmed the disappearance of crystals withdoi:10.1093/rheumatology/kep087 pmid:19447780 fatcat:47nana5bubdgtpmmor7yrm47ey