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Alteration of the Expression of A 2 a Adenosine Receptor and Toll-like Receptor 4 in Macrophage Cell Lines
2003
Japanese Journal of Oral Biology
Key words: A 2 a adenosine receptor/toll-like receptor 4/adenosine/ATP/LPS Abstract: Macrophages are essential for controlling the majority of infections, and are mediators of natural immunity. During an infection, lipopolysaccharide (LPS) stimulates macrophages to produce proinflammatory cytokines. Recently, it has been shown that A 2 a adenosine receptor (A 2 aR) is a critical part of the physiological negative feedback mechanism for the limitation and termination of tissue-specific and
doi:10.2330/joralbiosci1965.45.437
fatcat:w667qsxxzbfirea2rts7njzxzi