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autosomal dominant epilepsy with FS plus, generalized epilepsy with FS plus, and with severe myoclonic epilepsy in infancy that often begins with a prolonged FS. Various Na channels and GABAa receptors in the brain are probably involved in the pathogenesis of frequent FS and also, in simple FS. Genetic linkage analyses have mapped FS to four loci, FEB1,2,3 and 4, in chromosomes 8ql3, 19p, 2q23-q24, and 5ql4-ql5, respectively. (Hirose S, Mohney RP, Okada M et al. The genetics of febrile seizuresdoi:10.15844/pedneurbriefs-17-7-4 fatcat:3fqmzwl2hve33h5uhi7r47g7eq