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The deletion of the third exon of the growth hormone receptor (GHRd3) is one of the most common genomic structural variants in the human genome. This deletion has been linked to response to growth hormone, placenta size, birth weight, growth after birth, time of menarche, adult height, and longevity. However, its evolutionary history and the exact mechanisms through which it affects phenotypes remain unresolved. While the analysis of thousands of genomes suggests that this deletion was nearlydoi:10.1101/788653 fatcat:fabzqb4hjzdd7pf6u3rzkf5h7m