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Phosphorylation of the canonical histone H2A marks foci of damaged DNA in malaria parasite
[article]
2020
bioRxiv
pre-print
Plasmodium falciparum parasites proliferate within circulating red blood cells and are responsible for the deadliest form of human malaria. These parasites are exposed to numerous intrinsic and external sources that could cause DNA damage, therefore, they have evolved efficient mechanisms to protect their genome integrity and allow them to proliferate in such conditions. In higher eukaryotes, double strand breaks rapidly lead to phosphorylation of the core histone variant H2A.X which marks the
doi:10.1101/2020.11.06.372391
fatcat:el5lvhwpsnecxoiafpbiq3guy4