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In vivo administration of µ µ µ µ-opioid receptor selective agonists to various species is known to suppress lymphocyte, NK cell, and macrophage functions, in addition to mediate pain relief and euphoria. Using a mouse model in which the µ µ µ µ-opioid receptor gene was disrupted by targeted homologous recombination, we explored the involvement of this receptor in natural killer (NK) cell activity and T lymphocyte function. Following peripheral morphine administration, NK cell activity was notdoi:10.3844/ajisp.2006.35.39 fatcat:qlz3bjobpbbdnfshttv6qhzrja