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HMGB1 recruits natural killer cells by CXCL12/CXCR4 axis contributing to persistent airway inflammation and airway hyperresponsiveness during the late stage of respiratory syncytial virus infection
[post]
2022
unpublished
We previously showed that both high-mobility group box-1 (HMGB1) and natural killer (NK) cells contribute to respiratory syncytial virus (RSV)-induced persistent airway inflammation and airway hyperresponsiveness (AHR). Meanwhile, Chemokine (C-X-C motif) ligand 12 (CXCL12) and its specific receptor (chemokine receptor 4, CXCR4) were reported to play important roles in recruitment of immune cells. The relationships between them remains unclear. Here, we investigated whether HMGB1 recruits NK
doi:10.21203/rs.3.rs-1597705/v1
fatcat:7t6tjnxngfeptfjwtxtnsta7ae