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Osteoporosis is a complex human disease that results in increased susceptibility to fragility fractures. It can be phenotypically characterized using several traits, including bone mineral density, bone size, bone strength, and bone turnover markers. The identification of gene variants that contribute to osteoporosis phenotypes, or responses to therapy, can eventually help individualize the prognosis, treatment, and prevention of fractures and their adverse outcomes. Our previously publisheddoi:10.1210/er.2009-0032 pmid:20357209 pmcid:PMC3365849 fatcat:sv2hwzp22naerftn55ojqapg4m