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Vasculogenic mimicry (VM) plays an important role in human glioma progression and resistance to antiangiogenic therapy as a compensatory neovascularization mechanism in malignant tumors. Caveolin-1 (Cav-1) has been found to contribute to VM formation. However, it remains largely unknown whether Cav-1 expression correlates with VM in glioma. Methods: In this study, by performing immnunohistochemical staining of Cav-1 and CD31/Periodic acid-Schiff co-staining, we found that Cav-1 expression anddoi:10.21203/rs.2.13473/v1 fatcat:zkm65zdmsjgntpymgcucit3bla