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Rationale: CD73 (ecto-5´-nucleotidase) on immune cells is emerging as critical pathway and therapeutic target in cardiovascular and autoimmune disorders. Objective: Here we investigated the role of CD73 in post infarction inflammation, cardiac repair and remodelling in mice after reperfused myocardial infarction (50 min ischemia). Methods and Results: We found that compared to control mice a) cardiac function in CD73 -/mice more severely declined after infarction (systolic failure with enhanceddoi:10.1161/circresaha.113.300180 pmid:23720442 fatcat:7cvtjt435zd6vdrtrr7mjkwtle