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Studies on the pathogenesis of type 1 diabetes have mainly focused on the role of the immune system in the destruction of pancreatic ␤-cells. Lack of data on the cellular and molecular events at the ␤-cell level is caused by the inaccessibility of these cells during development of the disease. Indirect information has been collected from isolated rodent and human islet cell preparations that were exposed to cytotoxic conditions. This article reviews in vitro experiments that investigated thedoi:10.2337/diabetes.50.2007.s52 pmid:11272203 fatcat:uyl7d3jzjnhpfblvpucaapdpda