MOESM7 of p66ShcA functions as a contextual promoter of breast cancer metastasis

Kyle Lewis, Alex Kiepas, Jesse Hudson, Julien Senecal, Jacqueline Ha, Elena Voorand, Matthew Annis, Valerie Sabourin, Ryuhjin Ahn, Rachel Selva, Sébastien Tabariès, Brian Hsu (+8 others)
2020 Figshare  
Additional file 7: Figure S7. A p66ShcA-induced epithelial to mesenchymal transition is dispensable for metastatic progression in a luminal breast cancer model. Immunohistochemical analysis of vector control (VC), p66ShcA-WT and p66ShcA-S36A overexpressing NIC mammary tumors using (A) E-Cadherin and (B) Vimentin-specific antibodies. The data is shown as average tumor volume (mm3) ± SEM (n = 7 tumors/group). Representative images are shown. Statistical analysis was performed using a one-way Anova with a Tukey's multiple comparisons test (*P
doi:10.6084/m9.figshare.11626671 fatcat:vkmpgq2j4rhsvoiaw2omaefkki