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Cardiac diseases are the leading cause of deaths worldwide; however, to date, there has been limited progress in the development of therapeutic options for these conditions. Animal models have been the most extensively studied methods to recapitulate a wide variety of cardiac diseases, but these models exhibit species-specific differences in physiology, metabolism and genetics, which lead to inaccurate and unpredictable drug safety and efficacy results, resulting in drug attrition. Thedoi:10.3389/fcell.2021.655161 pmid:33869218 pmcid:PMC8049435 fatcat:rxod2sq4irdfnfzeqj76k4s7p4