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Patients with sickle cell disease (SCD) typically require transfusions with RBC components, which exposes them to numerous, possibly foreign antigens and potentially causes them to produce an antibody or antibodies to the antigens they lack. As transfusion of these patients increases, the likelihood that they will produce an initial antibody or additional antibodies increases. Once a clinically significant antibody is produced, units of RBCs that lack the associated antigen should bedoi:10.21307/immunohematology-2019-371 fatcat:3q5ze6aixvedrotwpubdhmglua