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17β-estradiol suppresses hyperoxia-induced apoptosis of oligodendrocytes through paired-immunoglobulin-like receptor B
2016
Molecular Medicine Reports
Hyperoxia is a high risk factor for neurodevelopmental disorders and can cause nerve cell death. 17β-Estradiol (E2) has been demonstrated as a neuroprotective agent. In the present study, the effect of hyperoxia on rat oligodendrocyte precursor cells (OPCs) in vivo and the neuroprotective effects of E2 on these cells were investigated. OPCs were treated with various concentrations of E2 and were harvested for reverse transcription-quantitiative polymerase chain reaction (RT-qPCR) analysis at
doi:10.3892/mmr.2016.4808
pmid:26821234
fatcat:6ql4stzy6zfqbdm5nxser3diyy