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Liver disease is associated with profound and characteristic changes in lipoprotein composition and metabolism. The most pronounced alterations are the Formation of lipoprotein-X in intra-and extrahepatic cholestasis, the decrease of apolipoproteins A-I and A-II and the increase of apolipoprotein E. These alterations impair the activities of both lipoprotein lipase and lecithin : cholesterol acyltransferase. They are also responsible for an abnormal receptor mediated uptake of the lipoproteinsdoi:10.1515/cclm.19220.127.116.111 fatcat:jt2odtqwqjckfhvgtf6mcsslzy