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Current advances in arthroscopic surgery have led to good outcomes for arthroscopic Bankart repair (ABR) for recurrent anterior shoulder dislocation. However, recent studies have reported recurrence rates of 4%-19% after ABR. In our survey conducted from February 2002 to December 2010, the post-ABR re-dislocation rate was 8.8%. In 2011, we began performing the ABR with open Bristow (B) procedure or Remplissage (R) procedure in patients with large glenoid or humeral head bone defects and indoi:10.2739/kurumemedj.ms664006 pmid:34690207 fatcat:s2ryj5yol5fqdfeiune23e4sxy
more »... nts who play collision sports. Therefore, the present study is the second series evaluating the incidence of re-dislocation and instability after recurrent anterior shoulder dislocation. Surgery was performed for 84 cases of shoulder instability from January 2011 to August 2017. After excluding 7 open surgeries, 6 reoperations, and 2 patients with multidirectional instability, telephone interviews were conducted with 69 patients. The average follow-up duration was 46.9 months (range, 13-92 months). ABR alone was performed 61 patients; the B procedure was added for 3 patients, and the R procedure was added for 5 patients. Telephone interviews were conducted with 61 patients. There were no cases of re-dislocation or reoperation. Four patients who underwent only ABR experienced postoperative instability, but not to the extent that their daily lives were affected. This study showed that the addition of R or B technique to ABR for recurrent anterior shoulder dislocation resulted in a 0% re-dislocation rate.
The standard surgical method for symptomatic submucosal tumors (SMTs) or tumors with unclear biological behavior is enucleation. Minimally invasive approaches are usually considered appropriate for surgical enucleation; thus, thoracoscopic and laparoscopic enucleation is performed widely and safely. However, it is sometimes difficult to enucleate large and complicated esophageal tumors using thoracoscopic surgery, and even if rare, there is the risk of requiring thoracotomy or esophagectomy. Indoi:10.1186/s40792-020-00854-5 pmid:32377803 fatcat:xmtayf3v7zg2fgryuca5ch2gnq
more »... the present case, we enucleated a large and complicated leiomyoma safely using a new combined method with endoscopic and thoracoscopic procedures. A 42-year-old woman presented to our hospital for a detailed examination of an abnormal finding in her health check-up chest X-ray images. She complained of upper abdominal pain after eating, and computed tomography revealed an esophageal tumor measuring 60 mm in length surrounding her lower thoracic esophagus. Esophagogastroduodenoscopy revealed a huge complicated SMT at the esophagogastric junction. Cytological examination with endoscopic ultrasound-guided fine-needle aspiration showed that the tumor was a leiomyoma. To enucleate this large and complicated esophageal SMT safely and without damaging the esophageal mucosa, we performed endoscopic and thoracoscopic procedures. We created a submucosal tunnel, endoscopically, and then performed thoracoscopic surgery to enucleate the tumor completely from the esophageal muscularis. Using these combined procedures, we were able to easily mobilize even a complicated tumor of this size from the mucosa and completed the surgery thoracoscopically without difficulty. As a result, the tumor was dissected safely with a minimal defect in the muscularis and without damaging the mucosa. Finally, we closed the defect in the esophageal muscularis with continuous sutures, thoracoscopically, and closed the entry of the submucosal tunnel using clips, endoscopically. Using these combined procedures, we safely enucleated a huge complicated esophageal SMT. The increased mobility of the tumor after creating the submucosal tunnel contributed to the minimal defect in the muscular layer and prevented injury to the esophageal mucosa, possibly leading to fewer postoperative complications such as esophageal stenosis and local infection.
High reliability will be required even in Internet of Things (IoT) communications in the near future for control purposes. To enhance reliability, both uplink (UL) and downlink (DL) communication quality must be considered. In this paper, we focus on the improvement of DL physical layer communication. To enhance the reliability of DL, the use of spatial diversity is a promising technique. We deal with two transmit diversity (TD) techniques, space-time block coding (STBC) and cyclic delaydoi:10.1587/comex.2020xbl0037 fatcat:utpo4e3jazcftkczobhpu3ra7m
more »... ty (CDD), which are evaluated by experimentations in real environments. From the results of the experiments, we confirmed that STBC and CDD could improve frame error ratio (FER). CDD can achieve almost the same performance as STBC by setting the amount of cyclic shift (ACS) appropriately. For example, in a non-line-of-sight (NLOS) area where shadowing happens due to hillock, FER could be improved from 5.11 × 10 −2 to 1.59 × 10 −2 in STBC and 1.00 × 10 −2 in CDD.
The Japanese Journal of Urology
A 67-year-old woman was hospitalized because of a month old case of gross hematuria. A retrograde pyelogram revealed a spece-occupying lesion in the left kidney. A left renal angiogram showed tumor vessels which were irregular and tortuous. A computed tomography revealed a large mass with central necrosis and a tumor thrombus in the left renal vein. The patient underwent left nephrectomy and radiotherapy. The histological diagnosis was nephroblastoma which is very rare in adults. Forty-sixdoi:10.5980/jpnjurol1928.79.10_1703 fatcat:geyjnljaxbf23cf426ncnr3qqi
more »... of nephroblastoma in adults ahve been reported in Japan.
This review summarizes recent progress in understanding the pathogenesis of IgG4-related disease (IgG4-RD), with a focus on fibrosis. Several studies reported that CD4+ T cells with cytotoxic activity promoted by the secretion of granzyme and perforin, cytotoxic CD4+ T cells (CD4+CTLs), and disease-specific activated B cells, infiltrated inflamed tissues and cooperated to induce tissue fibrosis in autoimmune fibrotic diseases such as IgG4-RD, systemic sclerosis, and fibrosing mediastinitis. Andoi:10.3390/immuno2010013 fatcat:sw5k5c7ub5ezpcrjydm7v7wrrq
more »... ccumulation of cells undergoing apoptotic cell death induced by CD4+CTLs and CD8+CTLs followed by macrophage-mediated clearing and finally tissue remodeling driven by cytokines released by CD4+CTLs, activated B cells, and M2 macrophages may contribute to the activation of fibroblasts and collagen production. In IgG4-RD, this process likely involves the apoptosis of non-immune, non-endothelial cells of mesenchymal origin and subsequent tissue remodeling. In summary, CD4+CTLs infiltrate affected tissues where they may cooperate with activated B cells, CD8+CTLs, and M2 macrophages, to induce apoptosis by secreting cytotoxic cytokines. These immune cells also drive fibrosis by secreting pro-fibrotic molecules in IgG4-RD.
In fact, our preliminary experiment indicated that D-aspartate inhibits glucagon secretion, supporting this possibility (Iharada and Moriyama, unpublished observation). ...doi:10.1248/bpb.30.1329 pmid:17603175 fatcat:pclt4fsjxvhnjdmpdkynjlcwsm
Characteristics of irradiation-induced trapping of hydrogen were investigated for quantitative evaluation of tritium retention in F82H steel. Before and after irradiation of 0.8-MeV 4 He or 0.3-MeV H ions, deuterium depth profiles near the surface of a disk sample were observed by nuclear reaction analysis under continuous exposure of deuterium plasma. One type of trap, with a trapping energy of 0.66 eV, was observed after each irradiation. The ratio of trap production rate to atomicdoi:10.1016/j.jnucmat.2013.04.057 fatcat:kzisacszqfem5jznxz2cafholq
more »... t was 0.0046 and 0.0014 for He and H irradiation, respectively. Annihilation occurred around 600 K for H irradiation but was not observed even at 691 K for He irradiation. Traps are likely to be interstitial-like sites associated with dislocation loops. This study also indicates that helium plays a role in inhibiting trap annihilation. In addition, the deuterium diffusion coefficient in non-irradiated F82H was determined by a time-lag permeation experiment.
The interaction between the cancer cells and the peritoneal mesothelial cells (PMCs) plays an important role in the peritoneal dissemination in several types of cancer. However, the role of PMCs in the peritoneal dissemination of pancreatic cancer remains unclear. In the present study, we investigated the interaction between the pancreatic cancer cells (PCCs) and the PMCs in the formation of peritoneal dissemination in vitro and in vivo. The tumor-stromal interaction of PCCs and PMCsdoi:10.3892/ijo.2016.3829 pmid:28035373 fatcat:zg36hcwkavdlnn7pcwoztwprwe
more »... ly enhanced their mobility and invasiveness and enhanced the proliferation and anoikis resistance of PCCs. In a 3D organotypic culture model of peritoneal dissemination, co-culture of PCCs and PMCs significantly increased the cells invading into the collagen gel layer compared with mono-culture of PCCs. PMCs pre-invaded into the collagen gel, remodeled collagen fibers, and increased parallel fiber orientation along the direction of cell invasion. In the tissues of peritoneal dissemination of the KPC (LSL-Kras G12D/+ ; LSL-Trp53 R172H/+ ;Pdx-1-Cre) transgenic mouse, the monolayer of PMCs was preserved in tumor-free areas, whereas PMCs around the invasive front of peritoneal dissemination proliferated and invaded into the muscle layer. In vivo, intraperitoneal injection of PCCs with PMCs significantly promoted peritoneal dissemination compared with PCCs alone. The present data suggest that the cancer-associated PMCs have important promoting roles in the peritoneal dissemination of PCCs. Therapy targeting cancer-associated PMCs may improve the prognosis of patients with pancreatic cancer.
An alternative source of mesenchymal stem cells has recently been discovered: dental pulp stem cells (DPSCs), including deciduous teeth, which can thus comprise potential tools for regenerative medicine. DPSCs derive from the neural crest and are normally implicated in dentin homeostasis. The clinical application of mesenchymal stem cells (MSCs) involving DPSCs contains various limitations, such as high cost, low safety, and cell handling issues, as well as invasive sample collectiondoi:10.3390/biomedicines10051049 fatcat:kzu47zm755fonobvxar5utbzfq
more »... Although MSCs implantation offers favorable outcomes on specific diseases, implanted MSCs cannot survive for a long period. It is thus considered that their mediated mechanism of action involves paracrine effects. It has been recently reported that secreted molecules in DPSCs-conditioned media (DPSC-CM) contain various trophic factors and cytokines and that DPSC-CM are effective in models of various diseases. In the current study, we focus on the characteristics of DPSC-CM and their therapeutic potential against various disorders.
. † † The author is with Police Info-Communications Research Center, National Police Academy, Fuchu-shi, 183-8558 Japan. a) E-mail: email@example.com DOI: 10.1587/transcom.E98.B.736 Fig. 2 2 Cognitive ... Masafumi ...doi:10.1587/transcom.e98.b.736 fatcat:cbtqsuavfreubkqbeokg4tr56i
Pancreatic stellate cells (PSCs) enhance the malignant behavior of pancreatic cancer by interacting with cancer cells and producing extracellular matrix (ECM). To date, several stroma-targeted therapies for pancreatic cancer have been attempted, but these therapies are still not in practical use. Integrins expressed in stromal cells are involved in fibrosis of several organs, as well as promoting tumor malignancy. We investigated whether CD51, also known as integrin αV, expressed in PSCs wasdoi:10.3892/ijo.2016.3374 pmid:26846197 fatcat:lzguawpijjfq5l4ofng6qgrhjm
more »... ociated with stromal formation of pancreatic cancer and enhancement of tumor malignancy. We also assessed the effects of suppression of CD51 in PSCs on pancreatic cancer. Immunohistochemistry for CD51 in resected pancreatic cancer tissues showed that high expression of CD51 in the tumor stroma was associated with lymph node metastasis (P=0.025), positive pathologic margin (P=0.025), and shorter patient survival times (P=0.043). Lentivirus-mediated short hairpin RNA knockdown of CD51 decreased the proliferation and migration of PSCs. Quantitative real-time polymerase chain reaction showed that expression levels of genes related with ECM and tumor-stromal interactions were decreased by CD51 knockdown in PSCs. In a co-implantation model of pancreatic cancer cells and PSCs, tumor growth in vivo was inhibited by CD51 knockdown in PSCs (P<0.05). We also found reduced tumor stroma and decreased proliferation of cancer cells in implanted cancer tissues with CD51-silenced PSCs (P<0.05). Our results showed that CD51 expression in pancreatic cancer stroma is associated with enhanced tumor malignancy and that CD51 may be a potential therapeutic target for pancreatic cancer.
Proceedings of The ITE Annual Convention
Transmission Characteristics of OFDM Field Pickup Unit
Transmission Characteristics of OFDM Field Pickup Unit
学会年次大会 ITE ' 95 ： 19951TE Annual Convention 峯 茎 耋 1972 為わ 5 ' 毋 α OFDM 変 調方 式 デ ィ ジ タ ル FPU の 伝 送特性 Transmission Characteristics of OFDM Field Pickup Unit 森 山繁樹 廊 土 田 健 一掌 中 原 俊 二 寧 斉 藤 正 典 韓 Shige1 【i Moriyama ... Kenichi Tsuchida Syunji Nakahara Masafumi Sako ＊ NHK 放送 技術 研 究所 NHK Science and Technical Research Laboratories 艸 ( 株 ) 次 世 代 デ ィ ジ タ ル テ レ ビ ジ ョ ン 放 送 シ ス テ ム 研 究 所 Advanced Digita1 Television Broadcasting ...doi:10.11485/tvac.31.0_273 fatcat:dgadu2iop5dxplinxfq6mhqugy
The Japanese Journal of Sarcoidosis and Other Granulomatous Disorders
〔解説〕 IgG4関連疾患の唾液腺病変における病態生理 IgG4関連疾患の唾液腺病変における病態生理 −免疫学的異常を中心に− 森山雅文,中村誠司 Masafumi Moriyama, Seiji Nakamura 【要旨】 IgG4関連疾患(IgG4-related disease: IgG4-RD)は,全身の諸臓器にIgG4陽性形質細胞の浸潤と高IgG4血症がみられる 特異な疾患である.近年になって ...doi:10.7878/jjsogd.35.55 fatcat:pihax2powfd2jiyj6ijhlltx4q
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