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Revision:
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+title = "ARTHUR AZEVEDO E A VISÃO SÓCIO-HISTÓRICA DO PAÍS"
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+issue = "26"
+publisher = "Universidade Federal do Maranhao"
+license_slug = "CC-BY-NC-ND"
+refs = []
+[[contribs]]
+index = 0
+raw_name = "José Dino Cavalcante"
+given_name = "José Dino"
+surname = "Cavalcante"
+role = "author"
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+[contribs.extra]
+seq = "first"
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+content = "<jats:p>Arthur Azevedo, nos seus epigramas, contos e nas revistas de ano, faz uma leitura bastante satírica das relações sociais no Brasil. Atento a tudo que acontecia no país, através da imprensa, da qual era leitor e colaborador assíduo, reproduz, com uma boa dose de sátira, os acontecimentos políticos e episódios familiares nos seus textos. O presente artigo pretende analisar como os escritos do escritor maranhense dialogam com os fatos e pequenos episódios do cotidiano do Rio de Janeiro e do Brasil, de modo geral. Para tanto, usaremos os seguintes teóricos: Friedrich Engels, Antônio Candido, Nicolau Sevecenko, L. Mallard, entre outros.xa0</jats:p>"
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+start = "2022-12-27T00:00:00Z"
Revision:
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Revision:
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+key = "B26"
+year = 2017
+container_name = "Am J Roentgenol"
+title = "Lung cancer risk associated with new solid nodules in the national lung screening trial"
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+[refs.extra]
+authors = [ "Pinsky",]
+doi = "10.2214/ajr.17.18252"
+volume = "209"
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+key = "B27"
+year = 2021
+container_name = "Thorax"
+title = "Lung cancer probability and clinical outcomes of baseline and new subsolid nodules detected on low-dose CT screening"
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+[refs.extra]
+authors = [ "Kim",]
+doi = "10.1136/thoraxjnl-2020-215107"
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+key = "B28"
+year = 2018
+container_name = "J Thorac Oncol"
+title = "New subsolid pulmonary nodules in lung cancer screening: The NELSON trial"
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+content = "<jats:sec><jats:title>Background</jats:title><jats:p>Screening for lung cancer with LDCT detects a large number of nodules. However, it is unclear whether nodule number influences lung cancer probability. This study aimed to acquire deeply insight into the distribution characteristics of nodule number in the Chinese population and to reveal the association between the nodule number and the probability of lung cancer (LC).</jats:p></jats:sec><jats:sec><jats:title>Methods</jats:title><jats:p>10,167 asymptomatic participants who underwent LDCT LC screening were collected. Noncalcified nodules larger than 4 mm were included. The nodule number per participant was determined. We defined five categories according to the number of nodules (based on nodule type and size): one, two, three, four, and more than four nodules. We stratified the nodules as groups A, B, and C and participants as Amax, Bmax, and Cmax groups, and explored the association between nodule number and the probability of LC on nodule and participant levels.</jats:p></jats:sec><jats:sec><jats:title>Results</jats:title><jats:p>97 participants were confirmed to have LC. The probabilities of LC were 49/1719, 22/689, 11/327, 6/166, and 9/175 in participants with one, two, three, four, and more than four nodules (p&gt;0.05), respectively. In the Bmax group, the probability of LC was significantly higher in participants with one nodule than those with &gt;4 nodules (p&lt;0.05), and the probability of LC showed a negative linear trend with increasing nodule numbers (p&lt;0.05). Based on the nodule-level analyses, in Group B, LC probability was significantly higher when participants had a solitary nodule than when they had &gt;4 nodules (p&lt;0.05).</jats:p></jats:sec><jats:sec><jats:title>Conclusion</jats:title><jats:p>LC probability does not significantly change with the number of nodules. However, when stratified by the nodule size, the effect of nodule number on LC probability was nodule-size dependent, and greater attention and active follow-up are required for solitary nodules especially SNs/solid component of PSNs measuring 6-15 mm or NSNs measuring 8-15 mm. Assessing the nodule number in conjunction with nodule size in baseline LDCT LC screening is considered beneficial.</jats:p></jats:sec>"
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\nМета написання статті полягає у розробці прогностичної моделі підготовки майбутніх учителів до здоров'язбережувальної діяльності в університетському освітньому середовищі.
\nПроаналізовано внесок учених у галузі педагогіки, психології, валеології, шкільної гігієни, фізичної культури та спорту в розробці теоретико-методологічних аспектів підготовки майбутніх учителів до здоров'язбережувальної діяльності в освітньому середовищі. Обґрунтовано необхідність оптимізації підготовки майбутніх учителів до професійної діяльності в освітньому середовищі,яка потребує компетентнісного підходу. Серед його складових чільне місце посідає здоров'язбережувальна компетентність. У зв'язку з цим категорія здоров'я стає об'єктом педагогічного впливу.
\nСформульовано базові категорії дослідження, які є основою його поняттєво- категоріального апарату, зокрема «компетентність», «здоров'ябережувальна компетентність», «здоров'ябережувальна компетентність майбутнього вчителя».
\nРозроблено модель формування здоров'язбережувальної компетентності майбутніх учителів до професійної діяльності в освітньому середовищі. Вона представлена як комплексна система взаємопов'язаних структурних елементів, що спрямовані на досягнення мети. Виокремлено такі структурні компоненти моделі: ціннісно-мотиваційний, когнітивний, операційно-діяльнісний, емоційно-вольовий. Визначено педагогічні умови формування здоров'язбережувальної компетентності майбутніх учителів та критерії ефективності.
\nФормування здоров'язбережувальної компетентності майбутніх учителів у закладах вищої освіти потребує ґрунтовних знань, умінь і навичок, особистісних якостей і здібностей та вміння їх реалізовувати в професійній діяльності в освітньому середовищі.</jats:p>"
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+container_name = "Annu Rev Pathol"
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+container_name = "Cancer Discovery"
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+[refs.extra]
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+container_name = "Transl Oncol"
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+[refs.extra]
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+container_name = "Ann Epidemiol."
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+[refs.extra]
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+container_name = "Adv Clin Exp Med"
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+[refs.extra]
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+doi = "10.17219/acem/62120"
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+key = "B38"
+year = 2014
+container_name = "Breast Cancer Res"
+title = "Biology of breast cancer in young women"
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+[refs.extra]
+authors = [ "Azim",]
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+volume = "16"
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+key = "B39"
+year = 2018
+container_name = "Front Immunol"
+title = "Interferon-Gamma at the Crossroads of Tumor Immune Surveillance or Evasion"
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+[refs.extra]
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+container_name = "Oncotarget"
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+[refs.extra]
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+[[refs]]
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+key = "B41"
+year = 2019
+container_name = "Front Immunol"
+title = "Vitamin D Controls Tumor Growth and CD8+ T Cell Infiltration in Breast Cancer"
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+[refs.extra]
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+content = "<jats:sec><jats:title>Introduction</jats:title><jats:p>Advanced age and obesity are independent risk and progression factors for triple negative breast cancer (TNBC), which presents significant public health concerns for the aging population and its increasing burden of obesity. Due to parallels between advanced age- and obesityrelated biology, particularly adipose inflammation, we hypothesized that advanced age and obesity each accelerate mammary tumor growth through convergent, and likely interactive, mechanisms.</jats:p></jats:sec><jats:sec><jats:title>Methods</jats:title><jats:p>To test this hypothesis, we orthotopically transplanted murine syngeneic TNBC cells into the mammary glands of young normoweight control (7 months), young diet-induced obese (DIO), aged normoweight control (17 months), and aged DIO female C57BL/6J mice.</jats:p></jats:sec><jats:sec><jats:title>Results</jats:title><jats:p>Here we report accelerated tumor growth in aged control and young DIO mice, compared with young controls. Transcriptional analyses revealed, with a few exceptions, overlapping patterns of mammary tumor inflammation and tumor immunosuppression in aged control mice and young DIO mice, relative to young controls. Moreover, aged control and young DIO tumors, compared with young controls, had reduced abundance ofcytotoxic CD8 T cells. Finally, DIO in advanced age exacerbated mammary tumor growth, inflammation and tumor immunosuppression.</jats:p></jats:sec><jats:sec><jats:title>Discussion</jats:title><jats:p>These findings demonstrate commonalities in the mechanisms driving TNBC in aged and obese mice, relative to young normoweight controls. Moreover, we found that advanced age and DIO interact to accelerate mammary tumor progression. Given the US population is getting older and more obese, age- and obesity-related biological differences will need to be considered when developing mechanism-based strategies for preventing or controlling breast cancer.</jats:p></jats:sec>"
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+title = "Determination of levels of heavy metals in some selected traditional medicinal plants in Southern Ethiopia"
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