2020-02-13
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Inflammatory networks cultivate cancer cell metastasis to the liver
published
| article-journal
doi:10.1080/15384101.2020.1728013
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2023-03-01
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Mechanisms of antigen-induced reversal of CNS inflammation in experimental demyelinating disease
published
| article-journal
doi:10.1126/sciadv.abo2810
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2023-08-11
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Systematic review of challenging issues in pathology of intraductal papillary mucinous neoplasms
published
| article-journal
doi:10.1016/j.pan.2023.08.002
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2024-05-02
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Supplementary Methods and Figures from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.25735919
|
2024-05-02
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Supplemental Table 3: Pattern 7 Ordered Genes from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.25735922
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2024-05-02
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Supplemental Table 3: Pattern 7 Ordered Genes from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.25735922.v1
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2024-05-02
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Supplemental Table 2: Pattern 1 Pathways from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.25735925.v1
|
2024-05-02
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Supplemental Table 1: Pancreas Gene Markers from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.25735931.v1
|
2024-05-02
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Data from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.c.7213937
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2024-07-07
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Multi-omic profiling of intraductal papillary neoplasms of the pancreas reveals distinct expression patterns and potential markers of progression
published
| post
doi:10.1101/2024.07.07.602385
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2024-04-08
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Transfer learning reveals cancer-associated fibroblasts are associated with epithelial-mesenchymal transition and inflammation in cancer cells in pancreatic ductal adenocarcinoma
published
| article-journal
doi:10.1158/0008-5472.can-23-1660
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2024-05-02
|
Supplementary Methods and Figures from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.25735919.v1
|
2024-05-02
|
Supplemental Table 1: Pancreas Gene Markers from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.25735931
|
2023-08-03
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Treatment Response in First-Line Metastatic Pancreatic Ductal Adenocarcinoma Is Stratified By a Composite Index of Tumor Proliferation and CD8 T-Cell Infiltration
published
| article-journal
doi:10.1158/1078-0432.ccr-23-0535
|
2024-05-02
|
Supplemental Table 2: Pattern 1 Pathways from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.25735925
|
2024-05-02
|
Data from Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial–Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Ductal Adenocarcinoma
unknown status
| post
| CC-BY
doi:10.1158/0008-5472.c.7213937.v1
|
2024-12-03
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Tumor-derived CCL2 drives tumor growth and immunosuppression in IDH1-mutant cholangiocarcinoma
published
| article-journal
doi:10.1097/hep.0000000000001185
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